日志
purpose: to understand
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cap degradation, T lymphocytes and activation of mast cells together increase threshing. Macrophages foam cells release free radicals, lipid oxidation product and proteolytic enzymes, degradation, make connective tissue thin fibrous cap. Atherosclerosis of mast cells can release a lot of chemotactic factors, inflammation, activation and particle residues, they can make mononuclear cells together and smooth muscle cell proliferation. These mast moncler cells through the release of trypsin and elk protease, activated macrophages plaques and smooth muscle cells produce MMPs, it may be triggered matrix-degrading, fibrous cap digestion and coronary artery plaque broken mechanism. Inflammatory cell activation in the ACS also play an important role, such as mononuclear cells and T lymphocytes activation between linked in some way, on the immune cell moncler jackets activation is connected, namely a cell activation can lead to another cell activation. Rich points PMNs WBC (nucleus of thrombosis) found obvious autolysis with NiaoJiMei (u, NiaoJiMei antibody and PA) - can counteract the most active receptor. The incubation PMNs contains plasma can generate free u-series, u - PA PA activity in animal models in human body thrombosis and detected moncler vest thrombosis. Purification and free crossean neutrophilic granulocyte can produce and PMNs equivalent activity, t-pa, elastase and organizations to produce less dissolved protease G, the role of circulation PMNs on endogenous thrombolytic plays an important role (27). 7 anti-inflammatory treatment to improve the prognosis of ACS evidence statins have important antiinflammatory effects (28,29). Pravastatin and west stand simvastatin atheromatous plaque can moncler coats reduce the macrophages content (30), simvastatin, and atorvastatin in vitro and vivo can reduce inflammation and inhibit endometrial tissue factor and MMPs expression (31). Because the statin can inhibit the expression of adhesion molecules, so the patient regardless of whether all ACS blood disorders are using statin drugs should (32). Low-dose lovastatin can make acute coronary event reduces 37%. Conversely, Cheap Moncler if the ACS patients stop statins, cardiac events will increase (33). Will ring that add oxygen enzyme - 2 (2) inhibitors Meloxicam COX - for the ACS can reduce risk 34,3 (5). Ridker etc (36) that aspirin can reduce the high level of risk of patients with AMI CRP levels. Antibiotics can reduce adverse outcomes of ACS (37). Anyhow, the Discount Moncler local inflammatory cells can generate and cytokines released, cytokines can activate and endothelial have adhesion and anticoagulation characteristics. Inflammatory cytokine can reduce matrix synthesis and matrix-degrading, thus contributing to the rupture. Finally, cytokines can enhance endothelial cells and macrophages, cause of endothelin synthesis of partial contraction vascular smooth muscle cell material increased. A lot of evidence supports the hypothesis that Moncler Outlet in the process of atherosclerosis (early formation, development and final lesions were thrombosis) inflammation which plays an important role. Clinical research affirmative inflammation in the ACS prediction and prognosis index of value. However, the detailed ACS inflammation mechanism is unclear, the inflammatory process is broken because of coronary artery plaque before or consequences? The ACS for potential whether inflammation composition Moncler Sale treatment target? Infection factors of ACS and coronary heart disease are pathogenic role? These are the problems to be solved.The study of n/med tuberculosis bacterium Dipasic resistanceThe paper] : purpose: to understand Dipasic drug-resistant strains are effective and drug inhibitory concentration. Methods: medium in susceptibility to join 10 different concentrations fight n/med tuberculosis medicaments, join tuberculosis bacterium fluid 10-3 mg.
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